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A study using kinase dead Ikk knock-in [ Ikk KA/KA ] mice, mimicking similar phenotypic characteristics as seen in APECED patients, demonstrated that autoreactive T-cells permit fungal infection

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Animal models show anti-anxiety-like and pain-reducing effects, probably running through GHK-Cus broad gene modulation and inflammation pathways rather than any direct hit on neuroreceptors

In vivo, oral ALC supplementation restored social preference and medial prefrontal cortex MBP expression in PWSI mice, whereas cuprizone co-administration abolished these effects, suggesting that the therapeutic effects of ALC are closely associated with its impact on myelination